| Properties | Information | |
|---|---|---|
| PhytoCAT-ID | PhytoCAT-1672 | |
| Phytochemical name or plant extracts | Phoenix Dan Cong tea | |
| PMID | 30574049 | |
| Literature evidence | CONCLUSIONS: Phoenix Dan Cong tea may act as a substitute for natural antioxidants and as a promising anticancer agent due to its protective effect on human health. | |
| IUPAC name | NA | |
| Phytochemicals’ class or type of plant extracts | Tea | |
| Source of phytochemicals or plant Extracts | Phoenix Dan Cong tea | |
| Geographical availability | China | |
| Plant parts | NA | |
| Other cancers | Breast cancer, Cervical cancer, Lung cancer, Liver cancer, Colon cancer | |
| Target gene or protein | NA | |
| Gene or Protein evidence | NA | |
| Target pathways | NA | |
| IC50 | 30.9 ± 5.55 μg/mL against MDA-MB-231 | |
| Potency | Phoenix Dan Cong tea may act as a substitute for natural antioxidants and as a promising anticancer agent due to its protective effect on human health. | |
| Cell line/ mice model | L02, WI38, MDA-MB-231, SW480, HepG2, HeLa | |
| Additional information | In addition, PDCe can significantly induce apoptosis of MDA-MB231 cells, mainly through the death-receptor-mediated extrinsic apoptotic pathway. Internalized PDCe can not only downregulate intracellular reactive oxygen species levels but also induce oxidative damage to mitochondria in MDA-MB231 cells. | |
| PubChem ID | NA | |
| Additional PMIDs | NA | |
| Additional sources of information | NA | |
| Safety | NA |